An update to that.
Jo Nova has a look-see:
“.....Something like this HAPE high altitude event seems to be occurring in Coronavirus patients due to the virus. Covid-19 binds to an enzyme involved in controlling the vasoconstriction. It targets and sticks to the ACE2 enzyme, stopping it working. Normally ACE1 raises angiotensin II and ACE2 lowers it. Angiotensin II raises blood pressure (ACE Inhibitors, the very common drugs against hypertension, act against ACE1 to lower blood pressure). So if the virus multiplies to the point it takes out all the ACE2, then there is nothing stopping Angiotensin from creating a vasoconstriction crisis (or a type of cytokine storm).
As the blood pressure builds it will force the fluids into the alveoli, flooding the lungs, dropping the oxygen levels in a vicious cycle. The high pressure may also increase blood clotting which could block flow, clogging up the blood vessels further. But the clotting problem may be due to antibodies against the virus.....”
Full article: http://joannenova.com.au/2020/04/urgent-new-medical-theory-on-coronavirus-hold-the-ventilators-stop-blood-clots-instead/
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